Infectious AIDS: Have We Been Misled?: The Fallacy of the HIV-AIDS Connection. Dieser Artikel ist nicht verfügbar.Infectious AIDS: Have We Been Misled?: The Fallacy of the HIV-AIDS Connection

Infectious AIDS is a collection of thirteen articles originally published in scientific journals, which call into question the dogma of infectious AIDS. With such thought-provoking chapters as "HIV is Not the Cause of AIDS" and "AIDS acquired by Drug Consumption and Other Non-contagious Risk Factors," Duesberg explores the correlation (but not causality) between HIV and AIDS. By challenging popular AIDS theory, Duesberg investigates fresh possibilities that can transform the study and treatment of the AIDS virus.

Peter H. Duesberg, Ph.D., is a professor of molecular and cell biology at the University of California, Berkeley.

In 1968-1970 he demonstrated that influenza virus has a segmented genome. This would explain its unique ability to form recombinants by reassortment of subgenomic segments. He isolated the first cancer gene through his work on retroviruses in 1970, and mapped the genetic structure of these viruses. This, and his subsequent work in the same field, resulted in his election to the National Academy of Sciences in 1986. He was also the recipient of a seven-year Outstanding Investigator Grant from the National Institutes of Health from 1985-1992.

On the basis of his experience with retroviruses, Duesberg has challenged the virus-AIDS hypothesis in the pages of Cancer Research, The Lancet, Proceedings of the National Academy of Sciences, Science, Nature, Genetica, Journal of AIDS, AIDS Forschung, Biomed. & Pharmacother., New Engl. J. Med., Chemical and Engineering News, Naturwissenschaften, Research in Immunology , Pharmacology & Therapeutics and the British Medical Journal. He has instead proposed the hypothesis that the various AIDS diseases are brought on by the long-term consumption of recreational drugs and anti-HIV drugs, such as the DNA chain terminator AZT, which is prescribed to prevent or treat AIDS.

Based on 30 years of studies on viral cancer, and over 15 years on cellular genes related to viral cancer- or oncogenes, now termed cellular oncogenes, the conclusion was reached that viral carcinogenesis is statistically negligible, and that the evidence for cellular oncogenes is inconclusive. Therefore, the hypothesis was advanced that aneuploidy, an abnormal number of chromosomes, rather than cellular oncogenes, is the cause of cancer. The hypothesis promises improvements in cancer prevention by eliminating substances that cause aneuploidy from food and drugs.