Perner florian (64 Ergebnisse)

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

Zustand: New.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

Zustand: New.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

Zustand: New.

Zustand: New.

Zustand: New.

Taschenbuch. Zustand: Neu. Regulation of the cell cycle phosphatase CDC25C in acute leukemia | Florian Perner | Taschenbuch | Englisch | 2025 | Our Knowledge Publishing | EAN 9786208902773 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. …

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

PAP. Zustand: New. New Book. Shipped from UK. Established seller since 2000.

Taschenbuch. Zustand: Neu. Régulation de la phosphatase du cycle cellulaire CDC25C dans la leucémie aiguë | Florian Perner | Taschenbuch | Französisch | 2025 | Editions Notre Savoir | EAN 9786208902780 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. …

Taschenbuch. Zustand: Neu. Regolazione della fosfatasi del ciclo cellulare CDC25C nella leucemia acuta | Florian Perner | Taschenbuch | Italienisch | 2025 | Edizioni Sapienza | EAN 9786208902803 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. …

Taschenbuch. Zustand: Neu. Regulação da fosfatase CDC25C do ciclo celular na leucemia aguda | Florian Perner | Taschenbuch | Portugiesisch | 2025 | Edições Nosso Conhecimento | EAN 9786208902810 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. …

Taschenbuch. Zustand: Neu. Regulacja fosfatazy cyklu komórkowego CDC25C w ostrej bia¿aczce | Florian Perner | Taschenbuch | Polnisch | 2025 | Wydawnictwo Nasza Wiedza | EAN 9786208902766 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu. …

Taschenbuch. Zustand: Neu. Regulation der Zellzyklus-Phosphatase CDC25C in der akuten Leukämie | Florian Perner | Taschenbuch | 80 S. | Deutsch | 2019 | Südwestdeutscher Verlag für Hochschulschriften | EAN 9786202323307 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu. …

Buch. Zustand: Neu. Druck auf Anfrage Neuware - Printed after ordering - Das Praxisbuch für die tägliche Diagnostik am MikroskopNeben den klinischen und radiologischen Untersuchungen kommt der histopathologischen Untersuchung von Lungengewebeproben eine entscheidungsrelevante Bedeutung zu. In diagnostisch schwierigen oder zweifelhaften Fällen kann durch die histologische Begutachtung betroffener Lungenareale eine Diagnosesicherung erzielt und therapeutisch relevante Differentialdiagnosen ausgeschlossen werden.Die Diagnostik nicht-neoplastischer Lungenerkrankungen ist oft eine große Herausforderung für den diagnostizierenden Pathologen. Unter den knappen Zeitressourcen aber auch angesichts der stetig zunehmenden Anzahl neu definierter bzw. weiter verfeinerter Entitäten, ist es kaum möglich, den Überblick zu behalten oder gar sämtliche wesentlichen Kriterien zur Diagnosestellung aus der Erinnerung abzurufen.Dieses Buch bietet eine kurze, übersichtliche Darstellung der einzelnen Krankheitsentitäten und der diagnostischen Kriterien für die Untersuchung von Lungengewebeproben.Es begleitet den Pathologen bei der täglichen Arbeit am Mikroskop und erläutert kurz und knapp, welche diagnostischen Kriterien im konkreten Fall greifen und welche Differentialdiagnosen zu beachten sind. Die Autoren legen Wert auf eine standardisierte und übersichtliche Darstellung der einzelnen Krankheitsentitäten. Fallstricken, die zunächst die Diagnose in die falsche Richtung lenken können, werden aufgezeigt.Algorithmen erlauben dem Leser, gezielt den korrekten diagnostischen Pfad einzuschlagen und zügig zur richtigen Diagnose zu finden.Ein einmaliges Praxishandbuch für die tägliche Diagnostik des Pathologen.…

Taschenbuch. Zustand: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled proliferation. Clinically, FLT3-ITD is associated with a poor prognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense. 64 pp. Englisch.…

Taschenbuch. Zustand: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware 68 pp. Spanisch.

Zustand: New. Print on Demand.

Zustand: New. Print on Demand.

Zustand: New. PRINT ON DEMAND.

Taschenbuch. Zustand: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled proliferation. Clinically, FLT3-ITD is associated with a poor prognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense.…

Taschenbuch. Zustand: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled proliferation. Clinically, FLT3-ITD is associated with a poor prognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 64 pp. Englisch.…

Taschenbuch. Zustand: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware 68 pp. Französisch.